Pathogens
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Mycobacterium презентация
Содержание
- 3. Mycobacterium tuberculosis Mycobacterium leprae (uncommon) Mycobacterium avium-intracellulaire Complex (MAC) or (M. avium) Important Human Pathogens
- 4. Lipid-Rich Cell Wall of Mycobacterium CMN Group:
- 5. Acid-Fast (Kinyoun) Stain of Mycobacterium NOTE: cord growth (serpentine arrangement) of virulent strains
- 6. Photochromogenic Mycobacterium kansasii on Middlebrook Agar NOTE:
- 7. Improved Mycobacterial Isolation Medium
- 8. Eight Week Growth of Mycobacterium tuberculosis on Lowenstein-Jensen Agar
- 9. Pathogenic Mycobacterium spp. BCG AIDS patients
- 10. Mycobacterial Clinical Syndromes
- 11. Diagram of a Granuloma
- 12. Laboratory Diagnosis of Mycobacterial Disease
- 13. Differential Characteristics of Commonly Isolated Mycobacterium spp.
- 15. Mycobacterium tuberculosis
- 16. Mycobacterium tuberculosis Infections
- 17. Incidence of Tuberculosis in USA
- 18. Mycobacterium tuberculosis Infections (cont.) BCG (bacille Calmette-Guerin)
- 19. Pneumonia Granuloma formation with fibrosis Caseous necrosis
- 20. PPD Tuberculosis Skin Test Criteria PPD = Purified Protein Derivative from M. tuberculosis
- 21. Chest X-Ray of Patient with Active Pulmonary Tuberculosis
- 22. Mycobacterium Tuberculosis Stained with Fluorescent Dye
- 24. Mycobacterium leprae
- 25. Mycobacterium leprae Infections
- 26. Mycobacterium leprae Infections (cont.)
- 27. Tuberculoid vs. Lepromatous Leprosy Clinical Manifestations and Immunogenicity
- 28. Lepromatous vs. Tuberculoid Leprosy
- 29. Lepromatous Leprosy (Early/Late Stages)
- 30. Lepromatous Leprosy Pre- and Post-Treatment
- 31. Clinical Progression of Leprosy
- 32. Effect of Cell-Mediated Immunity on Leprosy Clinical Outcome
- 34. Mycobacterium avium-intracellulaire Complex (MAC)
- 35. Mycobacterium avium-intracellulaire Infections
- 36. Mycobacterium avium-intracellulaire Infections
- 37. M. avium-intracellulaire Complex (MAC) Progression vs. CD4 Count in AIDS Patients
- 38. Mycobacterium avium-intracellulaire in Tissue Specimens Low Magnification High Magnification
- 40. REVIEW of Mycobacterium
- 41. Mycobacterium tuberculosis Mycobacterium leprae (uncommon) Mycobacterium avium-intracellulaire
- 42. Lipid-Rich Cell Wall of Mycobacterium CMN Group:
- 43. Pathogenic Mycobacterium spp. BCG AIDS patients REVIEW
- 44. Mycobacterial Clinical Syndromes REVIEW
- 45. Diagram of a Granuloma
- 46. Review of Mycobacterium tuberculosis
- 47. Mycobacterium tuberculosis Infections REVIEW
- 48. Mycobacterium tuberculosis Infections (cont.) BCG (bacille Calmette-Guerin)
- 49. Pneumonia Granuloma formation with fibrosis Caseous necrosis
- 50. Review of Mycobacterium leprae
- 51. Mycobacterium leprae Infections REVIEW
- 52. Mycobacterium leprae Infections (cont.) REVIEW
- 53. Lepromatous vs. Tuberculoid Leprosy REVIEW
- 54. Lepromatous Leprosy (Early/Late Stages) REVIEW
- 55. Clinical Progression of Leprosy REVIEW
- 56. Effect of Cell-Mediated Immunity on Leprosy Clinical Outcome REVIEW
- 57. Review of Mycobacterium avium-intracellulaire Complex (M. avium)
- 58. Mycobacterium avium-intracellulaire Infections REVIEW
- 59. Mycobacterium avium-intracellulaire Infections REVIEW
- 60. M. avium-intracellulaire Complex (MAC) Progression vs. CD4 Count in AIDS Patients REVIEW
Mycobacterium tuberculosis Mycobacterium leprae (uncommon) Mycobacterium avium-intracellulaire Complex (MAC) or (M. avium) Important Human Pathogens
Слайд 3Mycobacterium tuberculosis
Mycobacterium leprae (uncommon)
Mycobacterium avium-intracellulaire Complex (MAC) or (M. avium)
Important Human
Слайд 4Lipid-Rich Cell Wall of Mycobacterium
CMN Group: Unusual cell wall lipids (mycolic
acids,etc.)
(Purified Protein Derivative)
Слайд 5Acid-Fast (Kinyoun) Stain of Mycobacterium
NOTE: cord growth (serpentine arrangement) of virulent
strains
Слайд 6Photochromogenic Mycobacterium kansasii on Middlebrook Agar
NOTE: Mycobacteria pathogenic for humans can
be differentiated (Runyon Groups) by:
speed of growth (all are slower than most other pathogens) and by
production of chromogenic pigments (in light, in dark, or none)
speed of growth (all are slower than most other pathogens) and by
production of chromogenic pigments (in light, in dark, or none)
Слайд 11Diagram of a Granuloma
NOTE: ultimately a fibrin layer develops
around granuloma (fibrosis), further “walling off” the lesion.
Typical progression in pulmonary TB involves caseation, calcification and cavity formation.
Typical progression in pulmonary TB involves caseation, calcification and cavity formation.
Слайд 18Mycobacterium tuberculosis Infections (cont.)
BCG (bacille Calmette-Guerin) = attenuated M. bovis
Positive PPD
+ Chest X-Ray +
MDR-TB a serious global health threat
Слайд 19Pneumonia
Granuloma formation with fibrosis
Caseous necrosis
Tissue becomes dry & amorphous (resembling cheese)
Mixture
of protein & fat (assimilated very slowly)
Calcification
Ca++ salts deposited
Cavity formation
Center liquefies & empties into bronchi
Calcification
Ca++ salts deposited
Cavity formation
Center liquefies & empties into bronchi
Typical Progression of Pulmonary Tuberculosis
Слайд 41Mycobacterium tuberculosis
Mycobacterium leprae (uncommon)
Mycobacterium avium-intracellulaire Complex (MAC) or (M. avium)
Important Human
Pathogens
REVIEW
Слайд 42Lipid-Rich Cell Wall of Mycobacterium
CMN Group: Unusual cell wall lipids (mycolic
acids,etc.)
(Purified Protein Derivative)
REVIEW
Слайд 45Diagram of a Granuloma
NOTE: ultimately a fibrin layer develops
around granuloma (fibrosis), further “walling off” the lesion.
Typical progression in pulmonary TB involves caseation, calcification and cavity formation.
Typical progression in pulmonary TB involves caseation, calcification and cavity formation.
REVIEW
Слайд 48Mycobacterium tuberculosis Infections (cont.)
BCG (bacille Calmette-Guerin) = attenuated M. bovis
Positive PPD
+ Chest X-Ray +
MDR-TB a serious global health threat
REVIEW
Слайд 49Pneumonia
Granuloma formation with fibrosis
Caseous necrosis
Tissue becomes dry & amorphous (resembling cheese)
Mixture
of protein & fat (assimilated very slowly)
Calcification
Ca++ salts deposited
Cavity formation
Center liquefies & empties into bronchi
Calcification
Ca++ salts deposited
Cavity formation
Center liquefies & empties into bronchi
Typical Progression of Pulmonary Tuberculosis
REVIEW
